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: Unique E®: Unique E® Vitamin E is A.C. Grace Company's only product and has been since 1962. What makes Unique E unique in the world of Vitamin E?
Four Vital Functions of Vitamin E for a Healthy Heart The following quotes are taken
from a speech delivered by Dr. Evan Shute of the Shute Foundation
of London, Ontario, Canada, to show how Vitamin E acts in the body:
These four functions , all of them extensively confirmed in animal experimentation and human clinical work, make it the most valuable ally the cardiologist has yet found in the treatment of heart disease. It has no rivals. No other substance has this array of needful properties. This drug then becomes the first safe drug which can be given to patients suffering from the results of a clot in a coronary artery. There has been and still is no treatment at all for this type of case except two slightly effective drugs, which can be administered at great peril to the already precarious patient. Vitamin E replaces ‘rest and reassurance,’ which have no authentic basis, with real help to the damaged, laboring heart itself. It is the key both to the prevention and treatment of all those conditions in which a lack of blood supply due to thickened or blocked blood vessels or a lack of oxygen is a major part or the whole story of the disease. No pharmacologist or internist can suggest another substance with all the powers and properties of this vitamin. God made it unique and we ignore it at OUR peril.” Natural Vitamin E vs. Synthetic "Natural vitamin E supplements outperform synthetic forms, according to a VERIS Research Summary just published. Results of recent studies suggest that natural vitamin E is more bioavailable and is retained in body tissues significantly longer than synthetic vitamin E. These studies show that previously accepted differences were underestimated and that the bioavailability of natural vitamin E is about twice that of synthetic vitamin E compounds." TOWNSEND LETTER for DOCTORS & PATIENTS, JULY 1999, P. 100 Natural vitamin E is derived from vegetable oils, primarily soybean oil. Synthetic vitamin E is produced from petrochemicals. A key difference scientists are examining is the structure of the compounds. Natural vitamin E is a single stereoisomer while synthetic E is a mixture of eight stereoisomers - only one of which is the same as natural vitamin E. The other seven stereoisomers have different molecular configurations and lower biological activities. You can identify natural vitamin E by looking on the product's list of ingredients:
The researcher of a study that shows humans strongly discriminate between natural and synthetic vitamin E, believes anyone taking vitamin E should be taking the natural form. Our studies suggest without question that natural vitamin E delivers at least twice the impact as synthetic E, said Robert Acuff, PhD, Professor and Director, Center for Nutrition Research at East Tennessee State University. Dr. Acuff, author of a recent review examining more than 30 published studies on the differences between natural and synthetic forms of vitamin E, said In the case of vitamin E, the natural form is clearly the one our bodies were designed to use. Another researcher investigating the differences between natural and synthetic vitamin E found that the natural form is 100% more bio-available than synthetic. Maret Traber; Associate Professor at the Linus Pauling Institute, Oregon State University, in a recently published paper (Synthetic as compared with natural vitamin B is preferentially excreted as alpha-CEHC in human urine; studies using deuterated alpha-tocopherol acetates; Maret G. Traber et al. FEBS Letters 437 (1998) 145-148), wrote that the body seems to discriminate between the two forms, favoring retention of the natural form for use within the body and excretion of the synthetic form. This finding opens a new era in the natural versus synthetic discussion. While it has been acknowledged for some time that the body prefers natural vitamin E in terms of retention, this is the first evidence to suggest that the body preferentially excretes the synthetic form. In Dr Traber's study, six people were given 150 mg each of natural and synthetic vitamin E. As expected from previous studies, the researchers observed about twice the increase in plasma tocopherol concentrations following the administration of natural vitamin E as compared with the synthetic form. More significant, however were the differences they found in analyzing urine. Almost three times as much of a degradation product of alpha-tocopherol was observed in urine following ingestion of the synthetic vitamin E. This suggests that the natural form may be preferentially bound to the tocopherol-binding protein in the liver before being secreted into the circulation, whereas the synthetic vitamin E is metabolized at a higher rate and excreted. With numerous studies indicating that vitamin E supplements may help reduce the risks of age-related health conditions, research will continue to be published in this area. VERIS Research Information Service is a not-for-profit organization that strives to provide a responsible source of current research findings on the role of nutritional factors in health, with emphasis on vitamin E and other antioxidants, to health professionals, researchers, and health and nutrition educators/communicators worldwide. Introduction: A Comparison of Natural and Synthetic Vitamin E Vitamin E is the major chain-breaking antioxidant in body tissues and is considered the first line of defense against lipid perioxidation, protecting cell membranes at an early stage of free radical attack through its free radical scavenging activity Unless scavenged by an antioxidant, highly unstable free radicals attack the polyunsaturated fatty acids of cell membranes in a chain reaction. Extensive evidence implicates free radicals in the development of a number of degenerative diseases and conditions, including coronary heart disease, cancer; Alzheimer's disease, arthritis, premature aging and cataracts. Results of recent research suggest that vitamin B intakes that provide protection against degenerative conditions and chronic diseases may be substantially greater than amounts that prevent deficiency For example, results of a number of studies suggest that increased vitamin E intakes are associated with decreased risk of coronary head disease and certain types of cancer as well as enhancement of immune function. These increased vitamin B intakes (100 I.U. and above) are considerably above levels obtainable from diet alone, unless a very-high-fat diet is consumed, as most foods high in vitamin B are also high in fat. Most individuals who increase either intake of vitamin B to help prevent chronic diseases and maintain health choose supplements at levels that are considerably above recommended dietary allowances. Since supplements may contain either natural or synthetic vitamin E, the present VERIS Research Summary reviews the results of studies that compare the bioavailability of natural and synthetic vitamin E compounds. Vitamin E is the exception to the paradigm that natural and synthetic vitamins are equivalent because their molecular structures are identical. Natural vitamin E (RRR-alpha-tocopherol or d-alpha-tocopherol) is a single stereoisomer and is derived from vegetable oils, primarily soybean, sunflower and corn oils. Synthetic vitamin E (all-rac-alpha-tocopherol or dl-alpha tocopherol) is produced commercially by a chemical reaction of trimethylhydroquinone (TMHQ) with isophytol, resulting in a mixture of eight stereoisomers in equal amounts. Only one of the stereoisomers, constituting only 12.5% of the total mixture, is d-alpha-tocopherol. The other seven stereoisomers have different molecular configurations and biological activities that range from 21-90% of the activity of natural vitamin E based on rat fetal resorption tests. (Figure 1). In addition to the free alcohol form of vitamin E, acetate and succinate derivatives are often used in natural and synthetic vitamin E supplements. Vitamin E acetate and succinate esters are readily hydrolyzed in the digestive tract to the biologically active free tocopherol. Biological activities have been officially assigned to these compounds based on rat assays. (Figure 2) The bioavailability of natural forms of vitamin E is higher than that of synthetic forms, based on the animal assays and also demonstrated in human studies. recent human studies have shown that these differences are much higher than the officially accepted values. These studies have shown a strong discrimination between natural and synthetic vitamin E. It appears that this discrimination occurs after ab sorption from the digestive tract and that natural vitamin E is retained significantly longer in body tissues... Summary Studies suggest that vitamin E intakes considerably above recommended dietary allowances have beneficial effects in preventing a number of degenereative diseases and conditions. For individuals who choose to supplement their diets to increase their intake of vitamin E, a consideration of differences in bioavailability of natural and synthetic vitamin E compounds is relevant. A number of studies have demonstrated a strong discrimination between natural and synthetic vitamin E and that natural vitamin E is retained longer in body tissues. Recent research suggests that the previously accepted differences were underestimated and that the bioavailability of natural vitamin E is approximately twice that of synthetic vitamin E compounds... Correspondence: Forms of Vitamin E Many people are not aware that
there are several different forms of vitamin E. Why does the form
of vitamin E matter? Currently, physicians are limited to the following 4 choices [or various mixtures thereof] in dispensing or recommending Vitamin E in their practice:
The high quality diluent-free Unique E Complex Concentrate, providing full anti-thrombic and full anti-oxidant protection, costs up to 275% more than the industry standard vitamin E and saves money through effectiveness. Unique E is the only form providing full antithrombic (internal clot prevention) as well as full antioxidant (protection against harmful free radicals). Recent studies reveal the gamma tocopherol fraction is more potent than alpha tocopherol or the other beta and delta tocopherols in suppressing harmful free radicals. Gamma tocopherol inhibits peroxynitrates, harmful to brain cells because of its powerful oxidizing and nitrating properties. Vitamin E supplements should contain at least 20% gamma tocopherol, but most commercial Vitamin E supplements do not contain any gamma tocopherol at all. Unique E ®Vitamin E Complex has always contained at least 36% gamma tocopherol. Our Unique E® formula contains a minimum of 400 I.U. D-alpha-tocopherol, the important antithrombic factor, plus the antioxidant function d-beta, d-gamma, d-delta tocopherols. Q: What
Unique E® dosage should you take? Many physicians with long experience with Unique E® use a "jumpstart" maximum dosage of six Vegetable Gel capsules for quicker alleviation of chronic conditions, with the entire dosage all taken at the same time. Your doctor knows when the maximum dosage is advisable. When chronic symptoms are alleviated regular maintainence dosage based on body weight and age can be resumed. It is always advisable to follow your doctors directions in taking any vitamin or other supplement. There is no danger of overdose with Unique E® even with sustained maximum dosage. Q: How
long should you continue taking Unique E® Vitamin E?
Unlike the other oil soluable vitamins A, D, and K, Vitamin E contrary to general belief is not stored in the body. In as little as 3 to 5 days after discontinuing daily dosage, none is present in the blood stream and all preventive and protective benefits cease. According to nutritional authorities, the average American diet, currently provides only 7-10 I.U. of Vitamin E daily, an amount far too insignificant for any preventive or protective effect. For this reason the need for you to take Vitamin E continues as long as you desire its protective benefits. In insurance, you cease coverage protection when you cease payments regardless of how long you previously paid premiums. Q:
Can Vitamin E make you feel sick? If you are taking the synthetic dl type, you aren’t taking a natural substance. You are actually taking a synthetic chemicaly produced Vitamin E. When you take even low or normal dosages of the synthetic dl (or esterified) acetate or succinate forms, you may experience palpitations, gastric distress and other unwanted side effects even on relatively low dosages. This will not occur when taking the all natural unesterified high antithrombic high antioxidant form of Vitamin E Complex Mixed Tocopherols. Q: Why
is your doctor’s choice of Unique E ® Vitamin E Complex
best for you? Gamma Tocopherol Vitamin E in nature is a synergistic complex of Alpha, Beta, Gamma and Delta Tocopherols as well as Tocotrienols. The Tocotrienols are converted into the four Tocopherols, which are the active forms. The Natural Vitamin E Complex has two important functions in the body:
Gamma Tocopherol as the Anti-Oxidant Alpha Tocopherol serves as the antithrombic, while Gamma Tocopherol serves as the major factor in the antioxidant function. Beta and Delta have some antioxidant value. Alpha and Gamma serve two distinct functions: antithrombic and antioxidant.
Although Alpha and Gamma have two different functions in the body, neither functions correctly outside of the synergistic four part complex. This has lead to most of the misinformation about vitamin E as, since 1965, the industry standard has been the synthetic form. Most research on vitamin E as an antioxidant is uses the synthetic dl Alpha form, yet recent studies show that Gamma is a better antioxidant than Alpha Tocopheryl Acetate (or succinate). In
1965 the conventional "authorities" stated that Alpha Tocopherol
was the only active factor in vitamin E and that the Gamma, Beta,
Delta Tocopherols were useless. This claim supported sales of the
synthetic dl Alpha Tocopheryl acetate (or succinate). As Vitamin E
complex is researched, its healing properties are confirmed, such
as a 70% decreased risk of of Alzheimer's. Vitamin E Seen as Alzheimer's Foe! Wednesday, June 26, 2002 Eating plenty of foods rich in vitamin E may dramatically reduce the risk of developing Alzheimer's disease, according to the first population studies to rigorously examine the role that diet plays in protecting against the mind-robbing disorder. Reporting in a recent issue of the Journal of the American Medical Association, researchers from Rush-Presbyterian-St. Luke's Medical Center said subjects who consumed the highest levels of the antioxidant vitamin E reduced their risk of Alzheimer's disease by about 70 percent. In a separate JAMA article, scientists from the Erasmus Medical Center in the Netherlands found that a high vitamin E intake from food reduced Alzheimer's risk by slightly more than 40 percent. The Netherlands group, which followed 5,395 older people for six years, also found that smokers gained the highest level of protection with vitamin E and that vitamin C was somewhat protective against the disease, Dr. Marianne J. Englehart reported. The Rush team showed a weaker link with vitamin C. The vitamin E effect was seen in all age, sex and race categories, including people with heart disease, said epidemiologist Martha Clare Morris, who headed Rush's four-year follow-up study of 815 elderly residents in the Chicago Health and Aging Project. The Netherlands group, which followed 5,395 older people for six years, also found that smokers gained the highest level of protection with vitamin E and that vitamin C was somewhat protective against the disease, Dr. Marianne J. Englehart reported. The Rush team showed a weaker link with vitamin C. The vitamin E effect was seen in all age, sex and race categories, including people with heart disease, said epidemiologist Martha Clare Morris, who headed Rush's four-year follow-up study of 815 elderly residents in the Chicago Health and Aging Project. "There's very good evidence here that there may be some sort of protective effect from vitamin E," she said. "It's striking that two studies with different diets in two different countries with two different populations show the same findings that vitamin E intake from foods reduces the risk of newly developing Alzheimer's disease." 'Eat more vegetables' People in the study who gained the greatest protection basically followed the federal guidelines to eat at least five servings of fresh fruits and vegetables daily. "For a disease where it's been frustrating that we haven't been able to identify preventable risk factors, the idea that diet may be associated is very exciting," Morris said. "the message is clear: Eat more vegetables. It's something we can all do to protect against this devastating disease." Foods rich in vitamin E include vegetable and olive oils, nuts, seeds, whole grains, fortified cereals, green leafy vegetables such as kale, collard greens, turnip greens and spinach, and cantaloupe. The two studies support a growing body of evidence that antioxidants are an important defense against Alzheimer's disease and other major killer diseases. Antioxidants are naturally made in the body and can be consumed in foods. They neutralize free radicals, which are potentially dangerous molecular byproducts of metabolism. High fat and cholesterol diets increase free radical production, which can damage DNA and cellular membranes, as does cigarette smoking and excessive sun exposure. Healthy low-fat diets increase antioxidant protection. Autopsy studies of Alzheimer's
patients show extensive free radical damage to brain cells. Antioxidant
levels are also increased, and scientists believe it is an attempt
to combat the excessive free radicals, but apparently they are insufficient
to halt the disease process. Despite the positive findings, a JAMA editorial cautioned the studies rely on people recalling what they ate, and that further clinical trials that carefully monitor vitamin content of food are necessary to prove cause and effect. Role of supplements unclear The studies also did not answer the question of whether vitamin E supplements can reduce the risk of disease, primarily because fewer than one-fifth of the study subjects were taking supplements, and most started taking the supplement after the studies were underway. There was also concern that some
of those taking the supplements did so because they were already having
memory problems. Slowing the development of Alzheimer's disease would have a major impact on individual health and on medical costs. An estimated 4 million Americans have been diagnosed with Alzheimer's disease, and the current rate of 360,000 new cases each year is expected to triple in the next 40 years. "Even modestly effectively interventions that delay the onset of Alzheimer's disease by one to three years will substantially alleviate the growing economic and societal burden associated with this disease," said Daniel J. Foley, an NIA epidemiologist who co-wrote the JAMA editorial. Other supplement studies have already produced some promising results. High doses of vitamin E, for example, were able to slow the loss of mental function in patients diagnosed with moderate Alzheimer's disease, said Dr. Mary Sano of Columbia University's College of Physicians and Surgeons, who headed the study. Experts caution that although the vitamin E supplement studies appear promising, they are limited to individual vitamins. Healthy foods contain many vitamins and hundreds of antioxidants, many of which have not yet been identified. Angina Pectoris "The complete or nearly complete prevention of angina attacks is the usual and expected result of treatment with alpha tocopherol." - Dr. Wilfrid E. Shute, M. D., Cardiologist who treated over 30,000 heart patients during his career. Typically an attack of angina pectoris is described by the patient as a sensation of pressure, tightness, or heaviness behind the breastbone. It may become crushing in nature like a closing vise and may be very severe indeed. It can come on so suddenly that it seems instantaneous, with the victim suddenly subjected to unbearable pain that renders him unable even to walk. The attack can end quickly or can continue until it is treated. The pain has a tendency to radiate, usually to the left shoulder and arm all the way down to the fingers. It is often accompanied by great anxiety and a fear of impending death. A mild attack, however, may occur simply as a sensation of pressure within the chest. Characteristically, true angina pectoris is elicited when the oxygen demand is increased, which can occur because of excitement, exertion, or even after the eating of a heavy meal. However, intercostal pain may also be elicited by exertion, since an increased rate of respiration and greater movement of the chest may irritate the affected nerve, but it can also result from any change in position and frequently occurs while the patient is sitting in a chair or lying in bed, whereas true angina does not occur when the patient is at rest. Although it had long been supposed, because angina attacks are so frequently brought on by exercise, that the syndrome resulted from insufficient oxygen supply to the heart, it was first demonstrated in 1984 when Dr. Lawrence S. Cohen of Harvard Medical School and Peter Bent Brigham Hospital found an excess of lactic acid in the heart muscle during angina attacks. Since the consistently high quantities of lactate could only have been produced in the absence of oxygen, the findings represented a remarkably clean and uncomplicated demonstration of hypoxia, as reported to the Thirteenth Annual Convention of the American College of Cardiology in New Orleans. Since then it has gone almost unquestioned that angina is caused by oxygen lack, which knowledge should have provided the key to a treatment that would be more than palliative. The characteristics of such a treatment had become obvious. For example, it was possible for a paper in the New England Journal of Medicine (December 14, 1987, page 1,278) to commence with, "Because angina pectoris is a consequence of inadequate myocardial oxygenation, ideal therapy for this incapacitating symptom would be directed toward both increasing coronary blood flow and decreasing myocardial oxygen requirements. "The authors, Dr. Braunwald, Epstein, Click, Wechsler, and Braunwald, might just as well have gone on to name alpha tocopherol as their ideal therapy if these National Heart Institute doctors had only known it. It has already been pointed out in the chapter on coronary occlusion that alpha tocopherol simultaneously is a powerful fibrinolytic agent, with an action that causes arterial blood clots to disintegrate, and a vasodilator that will increase the blood supply to the heart by widening the arterial lumen. It also plays a third and equally significant role in its function, well known to food technicians everywhere, as an antioxidant. When it is pointed out that the consumption of polyunsaturated fats reduces the serum level of vitamin E and increases the likelihood of vitamin E deficiency, that is an expression of the consequence of vitamin E's antioxidant activity. Ordinarily the essential fatty acids released into the bloodstream by polyunsaturates are highly vulnerable to per-oxidation - linking their molecules one for one with molecules of oxygen. Vitamin E in the bloodstream, however, preferentially bonds with the fatty acids and prevents their oxidation. The vitamin E is destroyed in the course of this activity, which is why polyunsaturates in the diet in any quantity create a need for proportionately more of the vitamin. But by this antioxidant activity, the vitamin prevents oxygen from being converted into toxic peroxides, leaving the red cells of the blood more fully supplied with pure oxygen that the blood carries to the heart as well as other organs. By all these properties already existing in one drug - and - that drug virtually without side effects except through over dosage in very special cases - surely this drug, alpha tocopherol, is as well tailored to be the ideal therapy for angina as any material could possibly be. Unfortunately, alpha tocopherol remains largely unknown to the medical profession, and as a consequence the profession has wasted a frantic 20 years of search for a drug that already exists. Untold time and money have been spent in the development of amyl Nitrite, erythrityl tetranitrate, pentaerytlrritol Tetranitrate, and others in a long list of nitrates and nitrites, Iproniazid, and other monoamine-oxidase inhibitors. The result is usually a short-lived fad and a quick return to nitroglycerin, which has no therapeutic effect whatsoever, but is remarkably fast and effective in relieving an attack of angina. So after years and years of search for a drug that would treat angina, the choice available to today's doctor is still to use the brown nitroglycerin tablets that have a chocolate base or the white tablets with a sugar base. To the patient accustomed to walking around with a pocket full of nitroglycerin tablets and perhaps taking as many as 20 to 30 tablets a day, it could in no way represent a burden to take a few vitamin E capsules daily thus preventing the recurrence of angina attacks in most cases, and to be able to throw away the nitroglycerin. The complete or nearly complete prevention of angina attacks is the usual and expected result of treatment with alpha tocopherol. Angina patients are treated exactly like those who suffer the same symptoms upon recovery from a coronary occlusion. The results are comparable in every way, except that I have a vague impression that, on the whole, patients who have had a frank coronary occlusion respond somewhat better I don't know why. It is essential to recall that two investigators, after years of careful study of the available material and evidence, stated that in their opinion all patients who had developed angina pectoris had had a coronary occlusion, though usually of a small vessel so that the electrocardiographic changes were not diagnostic or were easily missed, if this is so, and I consider it probable, then angina represents a specific indication for preventive measures against the possibility of thrombosis. Unless there is some contraindication, such as hypertension, an angina patient is routinely started by me on 800 international units of alpha tocopherol a day and seen at intervals of six weeks for reassessment. If no result has been obtained within six weeks, the dosage is increased by 200 to 400 international units for the next six weeks. When we reach the dose on which their symptoms are relieved, it is continued permanently. [Dosage suggestion for Unique E is 400 IU for each 40 lbs. of body weight] Although the protection from a coronary thrombosis in these patients is very nearly universal, it sometimes happens that, after some years, the symptoms of angina recur. In such a case, it is necessary once again to increase the dose gradually and keep increasing it until the condition is relieved. In discussing the successful treatment for angina, however, it must be re-emphasized that a misdiagnosis can easily be made. There is one type of pain, which is very common in middle-aged or older people which is very commonly mistaken for angina pectoris. It has been given many different names, by many different authors, although recognized as a definite entity for years. Also, the site of the responsible lesion has been identified in different locations - the sterno-costal joint, the intercostal nerve itself, or the nerve root. To make matters more confusing, it can be on either side at any level and so the pain can be ascribed to referred pain from several different organs. The most common name for this condition is intercostal neuralgia or intercostal neuritis, and so the inference is that it is a definite lesion of the intercostal nerve. It is often referred to as a radiculitis, signifying a lesion in the nerve root. It has been called the rib syndrome. It has been often misdiagnosed as breast tumor, cholecystitis, etc. This pain nearly always occurs in the left upper chest in front in right-handed people and in the upper right chest in front in left-handed people. In fact, this is so usually the location and so typically right or left sided, that once the patient has described it you can confidently tell him whether he is right or left handed. Characteristic of this pain is that it occurs toward the front of the chest; but it is accompanied by marked tenderness between the ribs, and this tenderness can be followed all the way around in the intercostal space right to the lateral aspects of the spine. The patient may not be aware of the tenderness until the doctor puts pressure on the area. The author has seen hundreds of such cases, many of which have had the condition for years. There has been no very effective medical treatment available for this condition, and the author has developed his own theories and method of treatment which are very effective in nearly all - but not all - cases. The doctor of medicine has been taught for years that minor dislocations of joints or their immobilization in an abnormal part of their range of movement cannot occur. Therefore, the chiropractic and osteopathic approach to pain was considered necessarily based on incorrect theory and manipulation of no greater benefit than heat and massage. All this, too, has changed, and the president of the American College of Surgeons and, now, many others have attested to the value of correct manipulation in treating many joint lesions. The author has known this for 24 years. He discovered three osteopaths who could relieve chest pain of this type in most patients, often very dramatically. In one such case a man who couldn't walk 20 yards, without pain, after manipulation walked for miles up and down hill and through college grounds and climaxed it by running upstairs, where he arrived breathless and perspiring, but free of pain. However, again by chance, a most effective medical treatment for this condition was discovered. While investigating the possible usefulness of vitamin E ointment (30 I.U. alpha tocopherol per gram of petroleum jelly), Burgess and Pritchard of the Montreal General Hospital had demonstrated its usefulness in hastening the healing of indolent ulcers and in so doing had shown that the alpha tocopherol in the ointment was absorbed by the tissues under the skin right down to the periosteum of bone. It literally walks right through the skin as if it weren't there. Now we treat all such patients by the inunction of vitamin E ointment into the skin over the nerve root for ten minutes, followed by heat for another ten minutes to drive in still more alpha tocopherol This works miracles in one to three nights. If it doesn't work within this three-day interval, we send the patient to the osteopath - not just any osteopath, of course - and if this doesn't get the desired result, we use vitamin B, hypodermically and by mouth, but with little hope that it will be successful. Having excluded referred pain
from organs and having excluded intercostal nerve pain, presumably
pain that occurs in the chest on exertion or excitement (especially
after a heavy meal or soon after the oxygen reserve in the heart muscle
has been used up during sleep or after exertion just before elicitation
of pain) is due to coronary artery narrowing and resulting myocardial
anoxia. Like all our coronary patients, angina patients are urged to lead normal lives, as far as this is possible. We restrict them in only two ways. We ask that they don't try to show anyone how much they can lift or how fast they can run. Many patients have returned to hard manual laboring on farms and in factories without subsequent trouble. There is even a distinct possibility that such hard work does them good - as long as they are protected against sudden insufficiency by vitamin E saturation. It was reported by Doctors Smith and Kidera in Aerospace Medicine (38:742, July, 1967) that physical stress is beneficial in some cases of angina, because it helps the development of collateral circulation. A gradually extended exercise program leading up to an objective of jogging a mile in 20 minutes showed excellent results in 15 cases and poor results in six. The poor results, of course, were in dyspnea and early congestive failure. They could have been avoided. (from "Vitamin E for Ailing and Healthy Hearts," by Wilfrid E. Shute, M. D. with Harold J. Taub, Pyramid House, N. Y., 1969, pp 43-49). Scar Tissue Most extensive burns, as well as wounds in which large areas of skin are lost, are routinely grafted as soon as possible. This is a long and always painful process because of two characteristics of scar tissue: contraction and tenderness. In the healing process scar tissue contracts, leading to deformity and to some interference with the normal range of movement, particularly when the area involves underarms, groin, elbows, knees, or the skin of the neck. Also, scar tissue is tender and often painful to the touch. It sometimes forms excessively, becoming heaped up, red, and itchy. When vitamin E is used, any scars which do appear contract very little or not at all, and are not tender. Vitamin E prevents scar tissue from heaping up, or if that has already occurred, vitamin E will take away the redness and the itchiness. Often vitamin E diminishes old, unsightly scars. Vitamin E has a bacteriostatic (bacteria-limiting) action which is most useful in treating large burns and other large denuded areas, recent or chronic. For example, it allows the old, infected bases of chronic varicose ulcers to throw off the gross infection and, along with its oxygen-conservation abilities, vitamin E promotes the formation of healthy granulation tissue over which skin can grow and spread. Such healthy granulation tissue can sustain the life and growth of the skin cells falling on it from the surrounding intact skin surfaces. many such ulcers heal by this type of self-grafting. Vitamin E dilates small capillaries, speeding up their extension into granulation tissue, and into the edges of chronic ulcers. It also accelerates the opening up of collateral vessels around areas of major vessels, whether arteries or veins, that have become narrowed. . . Because vitamin E has the ability to decrease the need of oxygen to all cells, to prevent unwanted clotting in blood vessels of all sizes, to open up collateral channels of blood supply, and to directly benefit all collagen fibers, its beneficial effect on such skin lesions as psoriasis and varicose eczema is not surprising. Vitamin E cream or ointment will actually fill in acne or chicken pox scars on the face, often to the point where they are barely discernible. I know this as a father as well as in the role of clinician. While she was an undergraduate nurse, my daughter Karen had a very severe attack of chicken pox which left several scars on her face and forehead. Vitamin E ointment was rubbed in regularly and the scars disappeared entirely. My wife Dorothy suffered many cuts around the face from the shattered glass of the windshield in a car accident. Her face was so badly cut up and sore that she couldn't bear to wash it. Instead she applied vitamin E ointment liberally, and within a week her face was healed. There are no scars. Incidentally, I believe that vitamin E is a must for the after-care of all plastic surgery scars. I know that stretch marks on the abdomen that often follow pregnancy can be prevented or made much less obvious with vitamin E. Examples of the practical uses of vitamin E ointment, and of the oil squeezed out of capsules, keep increasing in the medical literature, as well as in lay publications. The direct application of vitamin E from the capsule is credited with clearing up such problems as plantar warts and itching scalp. Several mothers have reported that vitamin E ointment is their favorite remedy for diaper rash. One woman found that the ointment gave relief from a very uncomfortable chronic irritation caused by sweat and clothing under her large and pendulous breasts. The effect of vitamin E ointment on the common moles which may be present at birth or develop later, can be quite remarkable, but persistence in applying it at night is necessary. Sometimes moles simply drop off, leaving a smooth surface level with the skin. Journals Describe Response of More Serious Lesions The journals devoted to dermatology tell of a variety of skin lesions which respond to vitamin E therapy. Many of the contributions come from Ayers and Mihan of California, who explain how they became interested in vitamin E as a therapeutic agent: . . . Milton Stout presented before the Los Angeles Dermatological Society in 1950, a woman with pseudoxanthoma elasticum, whose cutaneous and visual impairment were restored to near-normal following administration of vitamin E for a period of one year. This astounding therapeutic accomplishment in a hitherto untreatable disease led us to carry out a continuing clinical investigation among our private office patients with some highly gratifying results. He lists the following as responding
to vitamin E: epidermolysis bullosa, Raynaud's phenomenon with gangrene,
scleroderma, calcinosis cutis, Darier's disease (in combination with
vitamin A), severe types of cutaneous vasculitis, subcorneal pustular
dermatosis, benign chronic pemphigus, and some cases of chronic ulcers,
discoid lupus erythematosus and granuloma annulare. Vitamin E Inserts The Canadian manufacturer of one of the best-known brands of Vitamin E was asked if it really was good for "piles." He telephoned us long distance to ask about vitamin E for hemorrhoids and about making a vitamin E suppository. My reply was that it could be useful because hemorrhoids were really varicose veins, and since vitamin E often does help varicose veins the suppository just might be a useful product. Evan doubted that vitamin E would be useful as a rectal suppository, but said he would like to see a vaginal suppository to use as a convenient way of treating altrophic vaginitis and other such conditions. He had been using vitamin E ointment to treat such problems and had found it very effective. Vitamin E suppositories -- known as "Vitamin E Inserts" -- have been on the market for years now and are really useful in both areas. Health Preserver: Defining the Versality of Vitamin E , by Dr. Wilfrid E. Shute, M. D., Rodale Press, Emmaus, PA, 1977, p. 3-4; 95-96. [Add MSM and Serrapeptase for additional scar reduction] Fibrocystic Breast Tumors "Indeed, Vitamin E's versatility has led to fresh discoveries and consequent reports in the medical journals and in the newspapers for the past several months. It relieves most cases of fibrocystic disease (breast lumps)." - from brochure titled "Overcome and Prevent Illness with Natural Vitamin E," by Dr. Wilfrid E. Shute, M. D., The Shute Foundation. Dr. William J. Mauer, D. O., an Osteopathic Physician in the suburbs of northwest Chicago learned about Unique E®, and was the first doctor in America to use it professionally. Dr. Mauer had such tremendous success in his practice with Unique E® that he stated to Roy Erickson, founder of A. C. Grace Company, "You have the Cadillac of Vitamin E. And you have no business withholding this from physicians.There are many complementary, alternative, integrative practicing physicians who treat heart disease by other means than surgery. And they need your product." Further, Dr. Mauer called and told us that he was having experiences with patients with scar tissue showing up on their EKG, due to previous heart attacks, surgery, etc. He said that within 6 months to 1 1/2 years, depending on how much scar tissue was present, he found that the last EKG showed no evidence of previous heart attack because the scar tissue had dissolved. Other physicians also reported in women with fibrocystic breast tumors, both breasts loaded, within 10-14 days on maximum dosage, the lumps disappeared. Others reported that painful leg cramps were disappearing within 10 days to 2 weeks, sometimes a little longer. Still others claimed severe angina often responded within the first few doses at maximum amount. Dr. Mauer also reported that his patients taking blood thinners were placed on the maximum dosage of Vitamin E and within 8-10 days he removed them from all blood thinners with no complications. He believes that Unique E® Vitamin E is not a blood thinner, never will be. He confirms that it prevents platelet aggregation and internal blood clumping. Though Vitamin E itself is not a blood thinner, it is just a blood anti thickener. So Dr. Mauer replaces the usual blood thinner drugs with the maximum dose of Vitamin E and tailors it according to the patient's needs thereafter. Painful Leg Cramps (Intermittent Claudication) "This condition is serious for several other reasons. First, if the patient survives long enough, the ailment will probably progress to gangrene and eventual amputation of one or both legs. . . "[In Haeger's experiment] among those not given vitamin E who survived (only half as many lived, compared with those in the vitamin E-treated group) there were eleven leg amputations, as against one in those who were given vitamin E." Dr. Wilfrid E. Shute, M.D., The Shute Institute The first symptom of intermittent claudication (the result of a hardening of the arteries in the legs) seems simple and rather unimportant. Typically, the atient has been walking for a while -- perhaps shopping, out for a stroll, or just back and forth to the job -- and suddenly he or she must stop because f a severe cramp in the calf of one leg. Standing still for a short time usually causes the cramp to go away. Then walking can be resumed. In time, the frequency and severity of cramps increase, while the distance that can be covered before the cramps occur, shortens. Eventually, both legs are affected and the patient is no longer ambulatory. This condition is serious for several other reasons. First, if the patient survives long enough, the ailment will probably progress to gangrene and eventual amputation of one or both legs. Second, the incidence of a heart attack or death from heart failure is greatly increased in these patients. Dr. Knüt Kaeger reported in Vascular Disease (5: 199, 1968) and in Prevention (March, 1975) on supervised double blind, cross-over trials of vitamin E therapy for these cases. He showed that such patients were nothelped ignificantly by vasodilators, anticoagulants (blood-thinning drugs), or multivitamin apsules that contain no vitamin E. However, they were helped to a significant degree by vitamin E therapy. Dr. Haeger thus confirmed previous work by Dr. A. M. Boyd, professor of surgery at the University of Manchester in England, reported in Lancet (2: 132, 1949) and Journal of Angiology (14: 198, 1963). Both Drs. Boyd and Haeger demonstrated that the patients treated with vitamin E urvived longer than any other group of claudication patients ever studied. Boyd used several x rays to show that the calcium, characteristically deposited in the walls of the arteries in advanced arteriosclerotic lesions, was slowly eliminated during the treatment. However, the most dramatic proof of the power of vitamin E was shown by Haeger's experiment. Among those not given vitamin E who survived (only half as many lived, compared ith those in the vitamin e-treated group) there were eleven leg amputations, as against one in those who were given vitamin E. It is likely that the one amputee who was getting vitamin E might well have saved his leg if this experiment had not been a double blind study, where neither the subjects nor the researchers knew who was getting the test substance. Otherwise, he would probably have been placed on an increased -- and probably effective -- dosage of vitamin E as soon as it was evident that his condition was not improving. Haeger also demonstrated that the blood supply is, in fact, increased in the legs of intermittent claudication patients treated with vitamin E. Health Preserver: Defining the Versality of Vitamin E , by Wilfrid E. Shute, M. D., Rodale Press, Emmaus, PA, 1977, p. 33-34. Blood Clots (Arterial Thrombi) Vitamin E, properly used, will dissolve the fresh clot without increasing the danger of embolism. It does this in dramatic fashion, usually in a matter of hours or days, with no residual damage to the return of blood from the leg. -- Dr. Wilfrid E. Shute, M. D., The Shute Institute As has been stated in the introduction, the leading cause of death in the civilized world today is thrombi forming in the coronary artery. It must be obvious that thrombi can form anywhere in the cardiovascular three and that the danger of embolism from such thrombi is, of course, always present. That thrombi in the veins of the extremities and pelvis are now very commonly found in puerperal and postoperative patients and in the chronically ill and bedridden and that the incidence of such thrombi had increased in almost parallel fashion to coronary thrombi is becoming evident. Similarly thrombosis of the cerebral arteries is becoming more common. In all cases of thrombosis, the treatment of choice is alpha tocopherol. In venous thrombosis, treated immediately the diagnosis is made, the results are spectacular; if treatment is begun later, however, it is less so. The vitamin's ability to dissolve fresh venous thrombi is well documented. In acute coronary thrombosis the direct action of the alpha tocopherol on the clot in the artery is greatly curtailed by the mechanical difficulty of getting the tocopherol to the clot because of the relative and absolute paucity of collateral vessels. Therefore, the clinical evidence of the effect of the drug depends upon the clinical effect on the patient and upon the serial changes in the electrocardiogram. In general terms, it can be stated that the electrocardiogram shows the same serial changes as occur in the typical case not treated with alpha tocopherol, but to a much lesser degree. However, the electrocardiogram recovery is much more rapid and much more complete than in the untreated patient. Clinically, the patient is usually much less ill, and has a quieter convalescence and an unusually rapid and complete recovery. We think the same to be probably true of the victim of cerebral thrombosis, but neither this nor the direct effect on the thrombus in the coronary artery is susceptible to proof and must, at least at present, remain a clinical impression. However, there is an interesting analogy between these two situations and the demonstrable effect of alpha tocopherol on burns. If alpha tocopherol ointment is applied to a burn immediately, only that tissue destroyed by the injurious agent is lost. Without alpha tocopherol ointment, the toxic products of the dead tissue destroy the injured but viable tissue beneath, and the burn depends. Similarly, in the coronary and cerebral accident the zone of injury and the zone of anoxia are invaded by the alpha tocopherol, and the infarct or area of softening is reduced. Recovery, therefore, is more rapid and complete. This is logical, and this we believe to be self-evident. Embolism is an ever-present danger and one that we have feared in treating coronary occlusions (from mural thrombi under the infarcted area) in our cases of thrombophlebitis and in our cases of chronic rheumatic heart disease with auricular fibrillation. We have had very, very few, indeed, to our great joy. I can remember but one embolism occurring in a coronary occlusion, and the pulmonary embolus resulting rapidly resolved with very little discomfort to the patient. There have been none in memory in cases of thrombophlebitis and several, but still relatively rare, emboli in chronic rheumatic heart disease with auricular fibrillation. Two cases of peripheral arterial embolism have been reported elsewhere. One, a riding or saddle thrombus occluding the common iliac arteries in both legs in a case of coronary occlusion, the other an embolus to the right common iliac artery in a patient with chronic rheumatic heart disease with auricular fibrillation. These cases certainly support our impression that the effect of the alpha tocopherol is directly upon the fresh thrombus itself, as has been demonstrated by several authors in the case of acute thrombophlebitis. Vitamin E for Ailing and Healthy Hearts , by Wilfrid E. Shute, M. D., with Harold J. Taub, Pyramid House, NY, 1969, p. 133-135. Diabetes Mellitus "By decreasing the need for more oxygen, preventing clotting in vessels, and opening up collateral circulation, the effects of the diabetic arterial changes can be halted and reversed even when the condition is severe. I mean severe even to the point where, for example, gangrene of the toes has developed. Very rarely is any additional tissue lost after vitamin E therapy is initiated." - Wilfrid Shute, M.D., The Shute Institute The great need for therapy in the treatment of diabetes is obvious. . . Laymen and doctors alike need to remember that the great discovery of insulin did nothing to decrease the incidence of diabetes mellitus. It prolongs the lives of many diabetics but does not prevent the degenerative changes in the walls of the arteries. Regardless of how well a diabetic's blood sugar is controlled, regardless of whether the diabetes is mild, moderate, or severe, all females and most males develop unusually rapid and severe hardening of the arteries, chiefly involving the vessels of the eyes, brain, kidneys, or extremities, or a combination of these. By decreasing the need for more oxygen, preventing clotting in vessels, and opening up collateral circulation, the effects of the diabetic arterial changes can be halted and reversed even when the condition is severe. I mean severe even to the point where, for example, gangrene of the toes has developed. Very rarely is any additional tissue lost after vitamin E therapy is initiated. Therefore, amputation of the involved limb above the knee (the usual procedure) is almost always avoided. Since we know that, in a diabetic, even the tissues in the terminal degrees of arteriosclerosis can be salvaged, it is logical to assume that the tissues affected to a lesser degree, perhaps not yet severe enough to cause symptoms, can also be reclaimed by vitamin E therapy, preferably reinforced with megavitamin C. One patient who had been a diabetic for many years and had marked hardening of the arteries in the retinas of both eyes with old and recenthemorrhages, had such deterioration of sight thathe had been forced to retire from work. On 1,600 IU of vitamin E a day, his eyesight gradually improved. He was able to return to his old job and carried on perfectly well with his correspondence and all other work. On one particular occasion this man had broken his glasses so he had made a date with his eye doctor as well as doctors at the Toronto clinic he frequently went to. At the clinic, for some reason, on that day they asked him all about his symptoms. When he said his eyesight had improved noticeably, they examined his eyes. The doctor looked into his eye grounds with the ophthalmoscope. The doctor then took off his glasses and wiped them, wiped the lens of the ophthalmoscope and looked at the man's retinas again. He called in his colleagues and they looked at the patient's eyes and asked him whathe had been doing. He told them he had been taking vitamin E. The patient left there and went to his eye doctor to get his glasses replaced. The eye doctor, of course, did an ophthalmoscopic examination and he did what the clinic doctor had done. He wiped his glasses and the lens of the ophthalmoscope and called in his partner for verification. He said he'd never seen anything like it before; it was just a beautiful disappearance of most of the signs of diabetic retinas. He confirmed the resolution of the hemorrhages and the decrease in the arteriosclerotic changes. One of the main dangers for diabetics
is physical traumatic injury that damages the tissues already low
in circulation and therefore low in oxygen and this can lead to gangrene
and eventually to amputation. We have had a great many diabetics who
had gangrene when they first came to us, but in each one all the tissue
ever lost was the tissue that was dead on the first day we saw the
patient. The infection and the gangrene had extended into her foot. (An English paper I once read said the gangrene beyond the base of the great toe always progresses to amputation above the knee.) We puther on vitamin E treatment with the result that she lost the affected great toe by self-amputation. Self-amputation merely means that the tissues which are still alive at the junction of the dead cells eat away the dead tissue and eventually a line of separation develops and the appendage, in this case the toe, drops off by itself. In some cases it is necessary to cut back the bone a little because it projects, buthealing is prompt and complete. This lady lost no more than her great toe and she walks perfectly well. She didn't have to go to a hospital and she didn't lose any money to hospitalization or drugs. She is still very much alive at age ninety or so, taking 1,600 IU of vitamin E per day. At the Shute Institute we have had patients from every state in the Union, including Hawaii and Alaska, every province in Canada and from the Isle of Capri, Switzerland, Ireland, England, Ceylon, Australia, and New Zealand. One patient was the first governor of Alaska when it achieved statehood, and the head of a university there. He was a diabetic so severely ill the first time he came to us thathe had a nurse with him who never left his side during the trip. He was going blind, he was developing
gangrene, as I remember it, he had hypertension and I think he had
had one heart attack. When I think of the slow acceptance of vitamin E, I like to remember that it took more than eight years for insulin to be accepted by establishment medicine. Eight years after it had been first used successfully on a human patient it was still not being used in the great Cleveland Clinic. A doctor friend of mine reported that the Cleveland Clinic's authority on diabetes insisted that insulin hadn't been proven effective, and that it hadn't been standardized. He considered life-saving insulin dangerous and refused to use it. Penicillin had similar difficulties in being recognized as a lifesaver. Now, I don't believe many, if any, doctors ever considered vitamin E dangerous, but plenty were, and are, skeptical of its value as a treatment. However, a great many doctors have become interested in vitamin E therapy and use it the same as it is used at the Shute Institute, and I have every reason to expect that acceptance to increase. Health Preserver: Defining the Versality of Vitamin E , by Wilfrid E. Shute, M. D., Rodale Press, Emmaus, PA, 1977, p. 64-67. Stroke Prevention A stroke can be major or minor. It can range all the way from the sudden fatal hemorrhage into and through the brain that is not too common, to the much commoner partial apoplexies. These latter can be very transient, even frequent, may cause no more than temporary weakness of a hand or the face or a foot -- or can be severe, involve paralysis of one half of the body, advance slowly and inexorably from very mild beginnings, and end in prolonged invalidism or in a quick fatality. The mild, transient strokes are probably due to spasm of the cerebral vessels, the severe and drastic ones to clots in vessels at the base of the brain. They all tend to recur if the patient survives his initial attack. They are usually associated with high blood pressure and hardening of the arteries. They may leave a good deal of residual paralysis and weakness -- or merely "childish" character changes, gradual loss of memory and the higher mental powers. Death chews at these people, playing cat-and-mouse with them. The prevention of hardening of the arteries is imperfectly understood. Diet may be a large factor in its causation, especially overindulgence in animal fats. The prevention and treatment of hypertension is more effective of late, and no doubt would be still better if alpha tocopherol were used for more cases of incipient nephritis and for more pregnancy toxemia's. But alpha tocopherol is most valuable here its power to treat and prevent thrombosis or clotting in the damaged blood-vessels. In these patients it is uniquely valuable. Even long after a "stroke" alpha tocopherol sometimes seems to improve the palsy very noticeably. Usually it fails, for the damage has been irreparable. But its occasional success justifies its trial. And it is the only safe insurance against further attacks of clotting in that old brain. Unfortunately, vitamin E never or only very rarely helps damage to brain function and personal character. We believe all those who have hypertension and fear a stroke, all whose who have had one and fear the usual recurrence, and all those who have residual paralysis after apoplexy, should be given alpha tocopherol. What else can one do? No one can suggest a good alternative. This is not to minimize the value of hypotensive drugs -- merely to add to the doctor's kit the most useful agent he has yet found when he faces these distressing patients. The Heart and Vitamin E, by Dr. Evan Shute, M. D., The Shute Foundation for Medical Research, London, Ontario, Canada, 1963, p. 73-74. Peripheral Circulation Thrombophlebitis A clot in the bloodstream can occur at any point in the body. Commonly it occurs in the extremities, but it can turn up in the eye, the throat, or anywhere. It seems to develop most frequently in the legs. I think that’s because those vessels are so far from the heart, and they are so long in the leg. A person stands and walks and the result is that the force of gravity can cause more trouble in the legs than in other places where the vessels are shorter and the blood goes through them at a better rate. Laymen tend to use the terms clot and phlebitis and thrombophlebitis interchangeably. There are very important differences. Phlebitis refers to an inflammation of the wall and a clot is present as well. Clots in the veins of the leg, the most common site of thrombophlebitis, can be very serious for two main reasons. During the acute phase, the whole clot, or part of it, may break loose inside the vein and be carried to and through the heart to be forced into the pulmonary arteries. If the clot is large enough, it may shut off all the blood supply to one or both lungs, or be forced into a branch of the pulmonary artery and shut off the blood supply to an area of the lung. This condition, called pulmonary embolism, carries a death rate of 50 percent. The second serious consequence of thrombophlebitis concerns the development of the clot in the veins with progressive fibrosis which eventually leads to the vein’s being obstructed at that point. When damage to a large vein is sufficiently extensive, the return blood flow decreases. Swelling and a sense of heaviness and aching in the leg result, and, in some cases where there is minor injury, chromic varicose ulcers develop. Phlebitis has a habit of recurring, and each recurrence takes its toll of available venous drainage, leading to an increase of signs and symptoms and the increased danger of ulceration. Vitamin E, properly used, will dissolve the fresh clot without increasing the danger of embolism. It does this in dramatic fashion, usually in a matter hours or days, with no residual damage to the return on blood from the leg. However, the tendency to recurrence of phlebitis must be controlled by a maintenance dose of vitamin E for the rest of the patient’s life. The response vitamin E brings in treating chronic phlebitis–the condition which arises in many of those who have already suffered an acute attack–is variable. The treatment is usually very much worthwhile, but may take several weeks to show its value in any given case. Some years after we’d been using vitamin E to treat thrombophlebitis, my brother Evan, who is an obstetrician and gynecologist, had a patient with a D & C in the hospital. On the day he went in to discharge her, he discovered that she had developed a tremendous thrombophlebitis in her thigh. Here was a chance to show other doctors what vitamin E could do, he thought. It was winter and the doctors entered that hospital through a cloakroom where thy left their overcoats and overshoes. They passed by a bulletin board there where any information of interest to doctors was posted. On the bulletin board my brother put a sign saying, "Mrs. Jones in room 000 had a D & C three days ago. I came in to discharge her today and she has a tremendous thrombophlebitis in her thigh, very visible and palpable. I’ve put her on 800 units of vitamin E and the thrombophlebitis will be gone in three days. This lady has given permission to any doctor to come and speak with her and examine her as the vitamin E treatment goes on. I repeat: the thrombophlebitis will be gone in three days, so I suggest that you go today." The next day he posted another note saying, "The thrombophlebitis is half gone, but the lady is still in the hospital and if you want to see her please see her today." On the third day he announced, "There is no evidence that this patient ever had thrombophlebitis, but she will be in the hospital of another day or two. If you want to talk with her about her experience with vitamin E, please visit her." It is interesting that only two doctors from that whole staff ever visited that patient. Even they visited by coincidence. Both of them happened to be passing by in the hall when my brother was coming out of the room. He grabbed them and said, "You’ve talked a lot about the values of vitamin E, now come and see for yourselves what it does." Remember, we’re discussing a cure for a major problem in surgery and medicine. Still my brother couldn’t arouse any interest in it among the doctors in a major hospital. A remarkable case involved a redheaded Irish woman. (For some reason, redheads are more likely to develop rheumatic heart disease or rheumatic fever than blonds, and blonds more likely that brunettes. I was taught that in medical school but I found it hard to believe until my experience proved it to be true.) This redheaded woman developed a very large clot in the varicose veins in her thigh. She had been Evan’s obstetrical patient and he prescribed 600 IU of vitamin E daily. The clot cleared up completely as far as anybody could tell within three and a half days. At her next pregnancy she developed thrombophlebitis in the same leg between the knee and the ankle and, again, with vitamin E, this cleared up in four and a half days. At every subsequent pregnancy (and this woman got pregnant once a year for the next five years), she developed phlebitis in that leg but on each occasion she put herself on vitamin E. She didn’t even bother telling Evan until her next visit when she would report another phlebitis which had cleared up in vitamin E. She should have been on a maintenance dose, but wouldn’t agree to it. Let my tell you about a nurse I treated, very obese, about sixty years old, who developed this tremendous phlebitis. The case was complicated by her suffering from both hypertension and rheumatic heart disease. Either of these complications requires careful dosage with vitamin E. Yet phlebitis calls for large dosages of E in a hurry. This was a true dilemma. I talked to the patient about it. I told her I planned to put her on 800 IU of vitamin E per day, and that such a dosage would ordinarily be too big for anyone who had rheumatic damage to her heart along with high blood pressure. I predicted that the phlebitis would be nearly gone in ten days but she would start to have shortness of breath, pounding of the heart, and general distress at about that time. I saw her then in ten days and she had developed the pounding and shortness of breath I predicted, and the phlebitis had almost gone. It had been there for some weeks before I saw her, so it wasn’t as simple as an acute one which can be cleared in three days. I stopped her vitamin E for a day and a half, then cut her down to a small dose and maintained her on that for about a week. Then I put her back to 800 IU knowing that once more she would get into trouble in ten days. Well, she did. But by this time the phlebitis had cleared up completely. Recently, I was prevailed upon to see one of my fellow villagers in Florida, a woman who was seriously ill with thrombophlebitis. She had gone to her doctor who proscribed anticoagulants and put her in the hospital. She had been in and out of the hospital twice and, over the course of months, saw no results whatsoever. Her legs were tremendously swollen. When I went to see her she was sitting in a chair with the most swollen and had so much phlebitis that it obviously involved the veins in her pelvis too. I put her on 1,600 IU of vitamin E a day. Three weeks after she went on the treatment the leg was nearly normal and the woman was driving her own car and doing her own shopping. When I saw her she was at a bridge party, looking fine, pain-free. Of course, one of the most widely reported cases of phlebitis was the one suffered by the former president, Richard M. Nixon. There’s no way of my knowing what caused his problem. I don’t know if he had any specific injury to his leg or if the psychological tension he was under was affecting his circulatory apparatus, or whether the phlebitis was jus spontaneous–as it can be. I do know that if I had seen Mr. Nixon when he first developed the thrombophlebitis I could have cleared it up in three days or less, by giving him 800 to 1,600 IU of vitamin E daily. My brother and I have done this hundreds of times in treating phlebitis. It’s hard for me to believe that none the important doctors in attendance know enough to treat ex-president Nixon with vitamin E. Now you can never recover completely from phlebitis and the disease does have a tendency to repeat when patients are treated in the orthodox manner. So it is most likely that Mr. Nixon will have another thrombophlebitis, and this time it could well be fatal. On the other hand, if Mr. Nixon were on a maintenance dosage of vitamin E of 800 IU daily, a recurrence would be highly unlikely, if not impossible. He could have been treated successfully in the first place, with no danger of the pulmonary embolism that developed. He would have had a speedy, uneventful cure in three or four days. No doubt about it, no argument. Health Preserver: Defining the Versality of Vitamin E , by Dr. Wilfrid E. Shute, M. D., Rodale Press, Emmaus, PA, 1977, p. 37-41. Overcome and Prevent Illness After using Vitamin E in the treatment of over 35,000 patients during the course of the last 40 years, I am still amazed at its power and versatility. It is now recognized as an essential vitamin whose role is to protect the integrity of the cell membrane and over half the cell's contents. It is, therefore, essential for the normal function of every cell, every tissue and every organ in the body. THERE is NO CONDITION, NORMAL OR ABNORMAL, IN WHICH VITAMIN E IS NOT DIRECTLY INVOLVED. It follows, therefore, that everyone needs to assess his intake and utilization of Vitamin E. The Vitamin E content in food has changed radically during this century. Even the best of diets now contain 5 to 10 international units of Vitamin E per day, compared to the average of 100 units at the turn of the century. Then, too, so few in even the more opulent populations have the best of diets, especially the teenagers, who should be laying a base for a future of optimum health and longevity. The very atmosphere in large towns and cities has changed, resulting in a need for Vitamin E as the most effective agent in protecting the lungs against pollution. For these and many other reasons, it is evident to those working with Vitamin E, that virtually everyone needs to supplement his intake of Vitamin E with at least 100 units a day. Vitamin E, at a much larger level of intake than is necessary for its vitamin effects, becomes not only a vitamin but a therapeutic agent; able to help the body overcome major illnesses an serious degenerative diseases. The therapeutic dosage level is approximately three to one hundred and fifty times what is considered to be its effective vitamin dosage range. It has not one, but many actions at this level. It is necessary to understand each of these actions in order to know why one substance exacts a curative or beneficial effect on so many illnesses and diseases which on the surface seem unrelated. Few substances have such a wide spectrum of useful actions; one, two or more of which may act to help the body overcome acute or chronic illness and/or degenerative conditions. For example, Vitamin E's ability to restore normal capillary permeability allows the body to return to normal function; the kidneys in acute nephritis and the heart in acute rheumatic fever . In addition, Vitamin E in cases of acute nephritis helps assure recovery by its anti-clotting actions; by decreasing the oxygen need of the kidney; and by its anti-inflammatory and anti-viral properties. The result is full recovery in 24 to 72 hours in most cases. Vitamin E, by these same actions, restores normalcy to the patient with acute rheumatic fever in a similar length of time, even resolving pleural and/or pericardial effusions. These specific actions of megavitamin E are documented many times over in the medical, biochemical and physiological journals; and in the many books available on the medical uses of Vitamin E. In general, it can be said that in any condition in which there is a deficiency of blood supply or a deficiency of available oxygen, Vitamin E will always help out in some degree, and many times will result in complete resolution or cure. The list of abnormalities that respond to Vitamin E therapy is constantly increasing. Many of these conditions are those considered and stated in medical texts and journals to be untreatable and incurable. For example, Dr. Abram Hoffer has treated an advanced case of Huntington's Chorea and returned him to apparent normalcy with a very large dosage of Vitamin E (4800 IU a day) along with Niacin. This disease involves nerve and muscle degeneration and wasting. It is said to be untreatable and invariably progresses to death, usually with severe mental deterioration to the point of insanity. Dr. Antonio J. DeLiz treated an advanced case of Crohn's Disease (severe ileum or colon inflammation and ulceration with bloody diarrhea), another incurable disease, with remarkable results using 16,000 IU of Vitamin E daily. I have been able to bring relief to several other individuals with Crohn's Disease who telephoned or wrote me after I lectured in Australia. Milroy 's Disease is another rare, incurable disease according to the medical texts and journals. This disease is due to poor development of the lymphatic system with a consequent chronic pooling of lymph in the legs evidenced by abnormal swelling. Added is the insult of recurring attacks of throm hophlebitis each of which adds damage to veins and increases the swelling. Every case I have treated has responded, and the early cases seem to be fully cured. An amazing record of success with the use of Vitamin E therapy in treating several skin conditions has been reported by dermatologists in the California area. Indeed, Vitamin E's versatility has led to fresh discoveries and consequent reports in the medical journals and in the newspapers for the past several months. It relieves most cases of fibrocystic disease (breast lumps). It is the answer, partially or completely, to most of the many complications of prematurity . These tiny infants enter the world before their tissues and organs are fully developed for life outside the mother's body. Often they must have oxygen supplementation to survive. However, this oxygen, while life saving may lead to serious complications such as blindness, lung engorgement and fibrosis (scar tissue), a serious form of anemia, and heart and liver damage. Vitamin E has proven very useful in combating all these conditions. Indeed, Canada's renowned pediatric hospital, THE HOSPITAL FOR SICK CHILDREN, has been using Vitamin E routinely for years in its premature ward. My older daughter, Barbara, is a speech therapist. She uses 800 IU a day on her hyperactive learning disability boys to calm them down and make them teachable. An incidental observation concerned the rapid, complete relief of eneuresis (bed wetting) in the younger brother of one of her patients. When Vitamin E was stopped, the lad began to wet the bed again. When Vitamin E was resumed, he once more regained control. Barbara had unexpected and exciting result from the use of 800 IU of Vitamin E given daily to a little girl who was unusually passive, not hyperactive, and who had been unable to learn in her public school class. These results suggest that Vitamin E may well normalize brain function, and might even increase intelligence. Vitamin E can make life more enjoyable in a variety of ways. Used on Plantar's Warts (warts on the bottom of the feet growing inward), it causes them to dry up and drop off, leaving virtually no scar, It fills in the ugly scars of acne and chicken pox, and the stretch marks of pregnancy It heals wounds without scar tissue contraction. It counteracts most of the pollutants in the air, minimizes damage caused by the side effects of many drugs, and combats several forms of industrial stress It helps boost the immune system when an illness puts stress upon it. Vitamin E is anti-infective, anti-viral and anti-fungal. It has cleared up several cases of fungus infection under the fingernails or toenails. It combats herpes simplex (cold sores on the lips) and since this has been published in dental journals, is now used by many dentists as a local application to the lips before the patient leaves the chair . By far the most important aspect of Vitamin E therapy, however, is still its role in treating diseases of the heart and blood vessels , Paul Dudley White said in 1965: "In fact, when I graduated from medical school in 1911, I had NEVER HEARD OF CORONARY THROMBOSIS, which is one of the chief threats to life in the U.S.A. and Canada today - an astonishing development in one's own lifetime! There can be no doubt but that coronary heart disease has reached epidemic proportions in the United States. where it is now responsible for more than 50% OF ALL DEATHS." (Consultant - June-July, 1965) This is an amazing statement coming from the best known and respected cardiologist of his day. He and Dr. Samuel Levine, who also graduated in 1911, were the first two cardiologists in America. Up to that time heart disease did not warrant specialization; it was just one of the many branches of internal medicine, Now cardiologists and heart surgeons are numerous and, as reported in U.S. NEWS AND WORLD REPORT for March 15, 1982, heart disease has become a 39 billion dollar ''business'', with some surgeons earning more than a million dollars a year. What Dr, White said, in effect, was that the number one killer in the civilized world today, did not exist or was so very rare that the "heart attack" properly called ''myocardial infarction," was not recognized until 1912 when Dr. Herrick described six cases. Reflect on the history of the
heart attack. From 1912 with six cases, it increased to the point
where it became the number one killer in the civilized world. It has
become the worst plague ever to hit mankind. In 1973, total deaths
from heart disease numbered 1,062,160, in the U.S.A. with similar
statistics in other civilized countries. The sad part of the story
is that during all these years, no one in ''The Establishment"
has been able to stop this rapid and steady increase or to modify
it in any way. Virtually every doctor accepted the low cholesterol diet as suggested by Dr. Ancel Keys. By 1979, it was obvious that this diet was useless, and the profession was told this by its acknowledged leaders. Now the prevailing theory - the only one left - is that the High Density Lipoprotein (HDL) part of the cholesterol protects against the risk of heart attack; and that the Low-Density Lipoprotein (LDL) part increases the risk of heart attack; and that triglycerides, discussed in many papers, may or may not be involved. If this theory holds, then every doctor and every cardiologist must give his patients vitamin E, since apart from the minor benefits of exercise, one cocktail a day, and being a female; the only way to really increase the HOL in those with a low initial level, while at the same time decreasing LDL and triglyceride level. is the daily intake of 600-800 IU of Vitamin E. Widespread use of Vitamin E by doctors is going to mean that the controversy over the coronary by-pass operation will finally be solved by its abandonment. There is so much evidence that the operation is dangerous and useless except as a placebo. Indeed, more patients are able to return to work and normal living when treated medically than those who submit-fed to surgery. NOW - consider what adding Vitamin E to these medical treatments will mean. Again, the real triumph of Vitamin E is now a matter of record. All of the ''authorities'' have indicated that nothing they have done is responsible for the half in the upward spiral of death and disability from heart attacks, or to the subsequent steady drop. It is estimated that 40,000 middle-aged American males due to die of heart attack in 1974 did not die. The decline has continued until the current deaths are 25 % less than 1974. This is due to the fact that one in seven Americans is now taking Vitamin E , and if you remember that it is mostly the older ones that have become aware of Vitamin E and its value, You wilI realize that Vitamin E, plus perhaps a major assist from Vitamin C and to a lesser degree Selenium, is totally responsible for this wonderful decrease in death and disability. Elsewhere, I have quoted several authorities who admit that nothing they have done could possibly be responsible. Vitamin E's success in the treatment of cases of myocardial infarction or ' 'heart attack" is chiefly due to three of its many actions. The attack is nearly always due to a clot forming in a branch of the coronary artery, shutting off blood supply to an area toward the apex of the heart. Vitamin E is the substance in the blood stream which prevents abnormal clotting so, when present in normal concentration, such a heart attack cannot occur. It given at the very onset of an attack, Vitamin E will dissolve fresh clots, minimizing or eliminating the death of the muscle deprived of its blood supply. Vitamin E also decreases the oxygen need of the heart muscle and so minimizes the extent of the damage, if some must occur. Vitamin E helps, therefore, to prevent the extension of damage; and to prevent fresh and recurrent infarctions, Slowly, it opens up collateral circulation and improves the health and function of the patient's heart in general. Of course, these same properties of Vitamin E act to improve the function of leg muscles with deficient blood supply and oxygen due to narrowing of the arteries that supply the leg. The classical double blind, crossover study of Knüt Haeger reveals the effectiveness of Vitamin E in improving limb function, but more particularly indicates the protection Vitamin E provides against gangrene, amputation and, in general, heart failure and heart attacks in these patients. Buerger's disease is a special type of blood deficiency to the extremities, both upper and lower, since it involves inflammation of the arterial lining and the complication of clotting of blood in the vessel. Vitamin E is effective in treating Buerger's disease. Vitamin E has proven to be of inestimable value in the treatment of Diabetes Mellitus (inability to properly metabolize carbohydrates due to insufficient insulin production). The administration of insulin to preserve the lives of patients, by reducing the symptoms of diabetes, is a miracle in itself. Unfortunately, those whose lives have been saved for years by insulin treatments face the increased risk of the complications of blood vessels narrowing and consequent tissue and organ lack of adequate oxygen. Strokes, heart attacks, blindness, high blood pressure, kidney disease and peripheral gangrene are hazards which every diabetic, especially women, face. Even when eyesight has decreased, kidney function has decreased and toes die; Vitamin E will rescue the majority of patients with a return of sight, with a return of kidney function, and with the saving of legs from amputation. Vitamin E is essential to the complete treatment of Diabetes Mellitus; as important as diet, as important as insulin. Possibly more people have been converted to the use of Vitamin E by its dramatic effect on burns of all kinds: fire, electrical, chemical, even sunburn. Vitamin E Ointment (30 IU of Vitamin E per gram) limits the depth and degree of the burns; usually preventing blistering, infection and toxic reaction from the killed cells. In severe sunburn and minor domestic burns, the results are unbelievable to the uninitiated. The value of Vitamin E in treating bed sores in the chronically ill, and in those with broken necks and consequent paralysis, has to be seen to be believed. Suffice to say that it is used routinely in an increasing number of convalescent and extended care hospitals and facilities. There are four conditions in which the remarkable effects of Vitamin E are rapid and dramatic obvious in a matter of days; acute nephritis, acute thrombophlebitis, acute rheumatic fever and congenital heart disease with cyanosis. Vitamin E works when the right product in the right concentration is given to the patient in the right way; with due regard to the substances which are antagonistic, namely estrogen, inorganic iron, and mineral oil, as well as those which are compatible, especially Vitamin C and Selenium, plus all the other vitamins. It is important to use a quality VitamIn E product in order to achieve the best results. I now use natural-source Vitamin E. Natural-source Vitamin E is more biologically active and therefore, probably twice as effective as a corresponding amount of the present synthetic Vitamin E. Natural-source Vitamin E is derived from vegetable oils and contains the isomer which occurs in nature. Synthetic-source Vitamin E is chemically synthesized from petroleum products and contains seven additional isomers which are not found in nature. The label copy of a Vitamin E product should identity it as being from a natural or synthetic source by the use of one of these terms:
Some Vitamin E products contain what is claimed on the label, but assay results for other products have found all or part synthetic Vitamin E, while only the more expensive natural Vitamin E is listed on their labels. REFERENCES:
Doctors Wilfrid and Evan Shute began the Shute Institute and Medical Clinic in the 1940's and are known for their extensive work with Vitamin E. Brochure "Overcome and Prevent Illness with Natural Vitamin E," by Dr. Wilfrid Shute, M. D., Cardiologist, The Shute Institute, London, Ontario, Canada. |
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